Preliminary scientific tests indicate conolidine could inhibit specific ion channels, lessening neuronal excitability and limiting agony signals. This mechanism is especially pertinent in neuropathic ache, in which abnormal signaling triggers persistent distress. Also, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to suffering perception. Computation... https://barryg206ljc7.blogitright.com/profile