Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our power to rescue SERT purpose in KI midbrain synaptoneurosomes with the inhibition of FAK suggests elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by amplified pFAK localization in five-HT https://pro3355544.answerblogs.com/32476750/the-definitive-guide-to-pro33